Dr. Jack Kruse
Sunlight has to sulfate cholesterol FIRST to get the Vitamin D train rolling in the skin. When it is sulfated it becomes water soluble in blood. When it isn't you upregulate LDL production. So taking D3 pills and living inside is raising your LDL.
Cholesterol and Vitamin D3 are nearly identical in chemical structure. Most people do not know this. Sunlight naturally sulfates these things (pic below). The only difference in both bio-molecules is a single double bond in the second ring of the cholesterol backbone. Remember light is BURIED at the electronic level in all things including cholesterol and Vitamin D. Your pills soaked in seed oil do not.
This gives Vitamin D3 one less hydrogen atom than the closed ring of cholesterol.
One hydrogen is the only difference at the atomic scale between them but there is a shit ton of energy buried in the bond biochemists do not see so they ignore it at your peril. Here you will see the wisdom of Tensegrity 6 play its role. Look it all the charge density sunlight adds to the system that a pill cannot.
Hydrogen is the ultimate chameleon for the sulfation and differentiation of cholesterol and Vitamin D3. What does it do? Vitamin D3 is synthesized from cholesterol esters in the skin upon exposure to specific frequencies of sunlight. When this happens and calcium becomes normal in the blood serum, both molecules remain sulfated by melatonin. This implies sunlight is a calcium channel blocker.
Melanin holds charge so it is also a calcium channel blocker. ACTH made from the POMC gene causes calcium to flow. It does the exact opposite of what other cleavage products of POMC do.
THIS HYDROGEN INTERACTION WITH and WITHIN THE SUN IS HUGE to maintain the sulfation of lipids and proteins. This is the basis of how a quantum dot battery is built in animals to create light from the metabolism that controls biochemicals in us.
It turns out how hydrogen acts chemically, depends wholly on the environment it is within. Does this means hydrogen can take different forms in our body if the environment of that region is controlled by information in some way? Is it a donor or a collector of electrons? Is it a metal or a gas? Science depends on compelling narratives, and few people seem to know the real story behind hydrogen. In fact, Nature has shown us how hydrogen can act as a metal or non-metal. Hydrogen makes life a cooperative quantum dance and it can make other elements do things they normally would not do.
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https://en.m.wikipedia.org/wiki/Lipofuscin
Lipofuscin appears to be the product of the oxidation of unsaturated fatty acids and may be symptomatic of membrane damage, or damage to mitochondria and lysosomes. Aside from a large lipid content, lipofuscin is known to contain sugars and metals, including mercury, aluminium, iron, copper and zinc.
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The composition of lipofuscin includes fats (20-50%), proteins (30-60%), a significant amount of carbon, nitrogen, phosphorus, sulfur, magnesium and aluminum. Fats are mostly represented by phospholipids (cephalin, lecithin, sphingomyelin), as well as cholesterol, triglycerides and products of peroxidation and polymerization of fatty acids. The most abundant amino acids in lipofuscin granules are glycine, valine, alanine, and proline.
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Lipofuscin Hypothesis of Alzheimer's Disease
https://pmc.ncbi.nlm.nih.gov/articles/PMC3235942/
Key Words: Aβ protein, Alzheimer's disease, Amyloid, Lipofuscin, Macular degeneration, Neurofibrillary tangles
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its amazing blue light makes Amyloid almost exactly like Prion does.
im just going to expect Dr Jack Kruse is right about most everything at this point.
Blue light spectrum
Biological effects of high-energy visible light
Lipofuscin
Dioxin
Fuscin
Furan
Heparan
Insulin
Plaque
Amyloid
Digoxin Toxicity
Cardiac Glycoside
all of the compound are CHONS, very specifically without Potassium.
Dioxin was the biggest clue, fire smoke (Dioxin) separated from the ashes (Lye Potash Potassium).
Functional Amyloid and Other Protein Fibers in the Biofilm Matrix
https://pmc.ncbi.nlm.nih.gov/articles/PMC6173796/
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Tubulin
Microtubule
Acetylation
Amyloid
Of microtubules and memory: implications for microtubule dynamics in dendrites and spines
https://pmc.ncbi.nlm.nih.gov/articles/PMC5221613/
Microtubule damage shapes the acetylation gradient
https://www.nature.com/articles/s41467-024-46379-5
Tubulin acetylation: responsible enzymes, biological functions and human diseases
https://pmc.ncbi.nlm.nih.gov/articles/PMC11113413/
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Cytoskeletal Drugs
Nocodazole
https://en.m.wikipedia.org/wiki/Cytoskeletal_drugs
https://en.m.wikipedia.org/wiki/Nocodazole
Cytoskeletal drugs are small molecules that interact with actin or tubulin.
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ATPase
Helicase
Tubulin
Microtubule
KATP
Potassium
SV40 T-antigen
Tubulin pools in human erythrocytes: altered distribution in hypertensive patients affects Na+, K+-ATPase activity